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While the biomarkers ALT and GGT have traditionally been viewed as being surrogate markers of liver fat [65] and may not be direct markers of histological improvement, emerging information suggests that liver fibrosis may be a dynamic process and that the rate of development of fibrosis disease is directly related to changes in disease activity [66]

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This combination data is largely preclinical and short-term and has not been validated in large human trials

additionally, other factors that influence PGE 2 production are also discussed