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Some studies have shown significantly lower activity of SOD in patients with generalised and localised vitiligo [15, 87, 113]

Our data analyses showed the deficiencies in GSH and dTTP with replication stress in WT-Bcr-Abl-32D cells as a contrast to high GSH and dTTP pools without replication stress in T315I-Bcr-Abl-32D cells

This allows EGCG to scavenge ROS and reactive nitrogen species (RNS), chelate labile iron and copper, inhibit pro-oxidant enzymes such as xanthine oxidase, and activate the Keap1-Nrf2-ARE pathway to enhance endogenous antioxidant defenses including HO-1, NQO1, and SOD2

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