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However, it is essential to note that sustained elevation in plasma glucose concentrations due to reduced pancreatic insulin release from chronic melatonin treatment, especially when extending beyond the sleeping period [140], may compromise melatonins potential to ameliorate AD pathology [141, 142]

Emerging evidence indicates that insufficient maternal folate levels during pregnancy may increase the risk of ASD in offspring, whereas folate supplementation has been associated with reduced neurotoxicity, enhanced neurodevelopment, and attenuation of ASD-related symptoms (Frye et al., 2018)