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For in vitro experiments, comparative analyses between different experimental groups were performed using t-student test and one-way ANOVA with Tukey post hoc tests for intergroup comparisons unless otherwise stated

ROS generation is controlled under physiological conditions but increases under pathological conditions, with high ROS levels potentially inducing carcinogenesis and oxidative stress-stimulated diseases

Preclinical studies indicate it counteracts toxin-induced liver injury by modulating oxidative stress and inflammation via pathways like Nrf2/HO-1 and NF-B

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