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Our results concluded that i) intracellular GSH is associated with antibiotic susceptibility, ii) extracellular GSH exhibited antibacterial activity regardless of antibiotic susceptibility and resistance, iii) the antibacterial activity of GSH synergistically enhanced susceptibility of conventional antibiotics, and iv) GSH-mediated acidity was partially associated with the antibacterial activity of GSH

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This leads to the release and cytoplasmic translocation of p53, which can subsequently activate transcription-independent pro-apoptotic signaling (Figures 6A,B)