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J Exp Med (2016) 213(11):2399412

Elevated blood TMAO levels cause an imbalance in cholesterol homeostasis which leads to influenced macrophages, which in turn causes foam cell development and atherosclerotic plaques [68, 69, 71]

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Some experimental models indicate that GHK-Cu may regulate fibrosis-related gene pathways, suggesting a possible role in conditions involving abnormal tissue scarring