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Hydroxyl radicals are extremely reactive and may cause extensive damage to cellular components

These alterations result in a significantly extended half-lifeup to 120 times longer than native IGF-1due to reduced binding affinity to insulin-like growth factor-binding proteins (IGFBPs)

Because our natural levels can be depleted by age and lifestyle factors, consistent daily support may help maintain optimal cellular function

Recent studies have identified multiple molecular targets and therapeutic strategies that inhibit ferroptosis, including iron chelators, lipid peroxidation inhibitors, and natural antioxidants capable of restoring GPX4 and GSH levels