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111 A remaining question is whether potential controlling factors including allosteric control of glycogen synthase (GS) and phosphorylases via glucose-6-phosphate (G6P), 112 in addition to the insulin-independent transport of glucose across the hepatocellular plasma membrane by GLUT2 (as a glucose-sensitive protein in liver cells), can impact hepatic glycogen metabolism

The dysregulated NAD + /NADH ratio, together with the absence of SIRT3, may cause cell cycle and mitosis arrest, and mediate metabolic changes in aging by activating AMPK to initiate senescence via the classical p53 pathway

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10.1016/j.jaci.2010.08.047 J