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WHO 2011 guidance on the use of HbA1c in the diagnosis of diabetes mellitus adopted in the UK and reflected in NICE NG28 and NICE diabetes pathways specifies that HbA1c should not be used for diagnosis when conditions affecting red blood cell turnover or haemoglobin are present

The postulated mechanism for causing such attacks is through stimulation of the porphyrin precursor, delta-aminolevulinic acid (ALA) Laboratory tests: Diclofenac prolongs platelet aggregation without affecting the bleeding time, plasma fibrinogen and factors V and VII to XII

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Phase I safety studies report good tolerability with no serious adverse events at tested doses [9]