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The increases in beta-hydroxybutyrate (78-181%) confirmed that the fatty acid oxidation mechanism was driving these changes at the cellular level

This would promote the generation of an electron bottleneck, favoring O 2 and H 2 O 2 formation (Gonalves et al., 2011)

Research in Thailand reported that the incidence of MetS in patients with CKD was 71.3%, displaying an evident higher incidence than that in patients without CKD ( 2.3 The pathogenesis of MetS-related nephropathy Insulin resistance is the critical feature of MetS, and insulin receptors are expressed in podocytes, tethered cells, renal microvascular endothelial cells, and tubular epithelial cells of the kidney ( 1 (TGF- 1 ) is increased in renal inflammation and renal fibrosis, which contributes to the progression of MetS-related nephropathy ( 1 could activate the tumor suppressor p53 in diabetic insults

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