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Other theories on the origin of endometriotic lesions include the embryonic rest theory (prenatal endometrial precursor cells differentiate and become established in the pelvic region) 5,6,7 , coelomic metaplasia (transformation of the peritoneal mesothelium), lymphovascular metastasis (transportation of endometrial cells via lymphatics or blood), or endometrial stem/progenitor cells (reviewed in 8,9,10 )

This mobile redox active arm can transfer electrons to the incoming oxidized Trx or internally to the Grx domain (C28/C31) of the partner subunit where reduction of oxidized glutathione (GSSG) occurs