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Clinical formulations typically incorporate appropriate antioxidant buffers or maintain GHK-Cu concentrations within therapeutic windows (0.1-10 M) to optimize beneficial effects while minimizing pro-oxidant risks

Although chemical reactions with mercaptans, proteins, or ascorbic acid in enzymes and/or biological mediators may initiate the release of NO from iron-sulfur NO complexes, light is reported to be the most common trigger [67]