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Patch-clamp electrophysiology of parvocellular neurons of the paraventricular nucleus also indicated that application of the CB1 antagonist AM251 eliminated ghrelins electrophysiological effects (Kola et al., 2008)

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In fact, several recent studies that have conducted genome wide searches for common genetic defects across large samples of ASD children have only identified rare de novo mutations, thereby pointing to acquired mutations and/or mutations secondary to errors in DNA maintenance rather than inherited genetic syndromes (11, 12)