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Notably, accumulating evidence suggests that both eryptosis and erythronecroptosis might be involved in the host defense against pathogens
Mechanistically, TMAO exacerbates IBD pathogenesis by modulating key signaling pathwaysincluding endoplasmic reticulum stress (ERS) via protein kinase RNA-like endoplasmic reticulum kinase (PERK) activation, NF-B-driven proinflammatory cytokine production, and NLRP3 inflammasome-mediated pyroptosisthereby disrupting intestinal epithelial integrity, amplifying macrophage and T cell activation, and skewing immune homeostasis toward a proinflammatory milieu
The most frequently reported side effects are nausea, vomiting, diarrhea, and constipation, driven by the combined effect both drugs have on slowing gastric emptying
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