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In summary, these results establish a nonmetabolic syndrome, immuneredox lipid-remodeling phenotype in IMDD that involves the NIMETOX pathway and contributes to the severity, stage, and progression of the disease [235, 236]

Thus, inflammasome regulation in wound healing is a dynamic interplay between host molecular sensors and microbial signals, where temporal control combined with a balanced microbiome determines whether wounds progress toward regeneration or chronic inflammation

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