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revealed that miRNA-17-92 exerted protective effects on endothelial cells by inhibiting erastin-induced ferroptosis via targeting the A20/ACSL4 pathway (Xiao et al., 2019a)

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The central clinical idea: when mitochondrial ATP production falls and oxidative stress rises, the brain may lose the energy and antioxidant reserve needed to regulate neurotransmission, maintain cell membranes, recycle methionine, produce SAM, clear adenosine and homocysteine, build glutathione and tolerate medications or supplements

Interdependence of lipoxin A4 and heme-oxygenase in counter-regulating inflammation during corneal wound healing