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In summary, aortic cross-clamping recapitulates some key components of the atrial phenotype in poAF patients, like atrial inflammation and Ca 2+ -handling abnormalities (148, 157), suggesting that atrial inflammation and Ca 2+ -driven atrial ectopy act in concert to produce poAF (152), whereas the abbreviation of APD and ERP reported in these animals models is not a typical feature of patients developing poAF (148, 156, 157)

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