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The innate immune system interacts closely with basic cellular processes such as autophagy and reactive oxygen species (ROS) [15], [16], thereby coordinating these cellular processes with the innate immune response

The synergy between exogenous MOTS-c and exercise is not merely additive: because MOTS-c mimics one of the molecular consequences of high-intensity exercise (AMPK activation via AICAR accumulation), concurrent exercise training may amplify the peptide's effects on GLUT4 translocation and mitochondrial biogenesis

The disease is heterogeneous and can be caused by different aspects such as microbial products, environmental triggers, or genetics

[193] Gundacker C, Komarnicki G, Zdl B, Forster C, Schuster E, Wittmann K