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It will be important to further investigate the role of HO-1 mediated mitochondrial iron overload in cardiac ferroptosis in vivo and the physiological implications of this mechanism in the pathogenesis of oxidative stress-induced heart disease

Similarly, lncRNA MEG3 mitigates chondrocyte ferroptosis through a competing endogenous RNA (ceRNA) mechanism, sponging miR-885-5p to enhance the expression of SLC7A11, thereby reducing lipid peroxidation and iron-dependent cell death induced by erastin (177)