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Notably, whereas PPAR agonism increases plasma HDL in humans, this is not the case in most applied rat and mouse models, as they lack the PPAR response element in the promotor region of ApoA1 (the major apolipoprotein of HDL)

Laboratory studies using 5-amino-1MQ in aged animal models have revealed that NNMT inhibition modulates many age-associated metabolic parameters, including alterations in muscle tissue metabolism and cellular stress response pathways