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By linking NET deposition to both direct parenchymal injury and local ischemia, this model explains how NETosis can amplify HIF-1 and NF-B activation in granulosa cells and thus perpetuate a self-sustaining inflammatoryhypoxic circuit in the PCOS follicular niche

Aug 2012;22(8):1235-1242

Cytochrome P450 expression and activities in human tongue cells and their modulation by green tea extract

As indicated in Table 1, numerous experiments have demonstrated that bacteria can transition to their L-forms in the presence of antibiotics, significantly diminishing their sensitivity to these drugs [7]