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References Jelovac, N., Sikiri, P., Rucman, R., Petek, M., Perovi, D., Konjevoda, P., Marovi, A., Seiwerth, S., Grabarevi, Z., Sumajstorci, J., Dodig, G., & Peri, J

Abnormal Treg/Th17 balance and heightened Toll-like receptor signaling have been described in both conditions, suggesting that similar immune architectures may support disease persistence once tolerance has been breached (48, 49)

PMID: 38191086 Chem Biol Interact Fatostatin inhibits SREBP2-mediated cholesterol uptake via LDLR against selective estrogen receptor modulator-induced hepatic lipid accumulation

Reactive oxygen species (ROS), in excess, can cause disruption to normal cellular function and can damage cells (in particular mitochondria, & DNA of the sperm and egg) and tissues