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Furthermore, upregulation of PFKFB3 was shown to be dependent on TGF-1-activated Smad2/3, as both blocking the TGF-1 receptor and silencing Smad2/3 abolished the induction of PFKFB3

Integration of data on dietary intake, physical activity, sleep, anthropometric measurements, and gut microbiota using machine learning algorithm predicted postprandial glycemic response to real-life meals, this enables personalized diets to reduce postprandial blood glucose (Zeevi et al., 2015)

The crosstalk between mitochondrial quality control and metal-dependent cell death

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