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Compared with GPX1-3, GPX4 is also involved in the reduction of complex membrane-associated phospholipid hydroperoxides such as phosphatidylcholine hydroperoxide and cholesterol hydroperoxide, in addition to reacting with soluble, low molecular weight hydroperoxides such as t-butyl hydroperoxide and cumene hydroperoxide [68]

Google Scholar Yang A, Palmer AA, de Wit H

Additionally, ROS promote arrhythmias by disrupting potassium channel function, inducing endoplasmic reticulum and mitochondrial dysfunction, and causing electrical conduction disturbances between endothelial cells and cardiomyocytes [329]