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PI3K specific blockade to target these cells and improve tumor outcomes has been widely studied in recent years with propitious outcomes in the preclinical stage

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More mechanistic work shows that intravenously delivered AD-MSCs blunt granulosa-cell apoptosis, curtail senescence markers (SA--gal) and normalise FSH and AMH by suppressing over-active PI3K/AKT/mTOR signalling in acute and chronic cyclophosphamide rat models, highlighting a conserved anti-ageing pathway [177]