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Its unique worm-like structure enhances drug release, pharmacokinetics, and tumor targeting

We emphasize its combined effect on hippocampal neurons, which are known to be particularly vulnerable (Iwasaki et al., 2003), counteracting both early and delayed neural damage and achieving full functional recovery (based on the Morris water maze test, inclined beamwalking test, and lateral push test)

No Rebound Hunger or Metabolic Adaptation Appetite suppressants that work through central mechanisms (GLP-1 agonists, sympathomimetics, serotonergic agents) typically create tolerance over time, requiring dose escalation, and often produce rebound hunger when discontinued

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