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The PFVYLI-CAND peptide, which includes a hydrophobic CPP sequence, showed no efficacy in inducing cell death in either cell type (Figure

The acetylation signal was increased upon addition of acetyl-CoA, suggesting SnCE1 uses acetyl-CoA as a donor molecule for an enzymatically catalysed autoacetylation

Second, given that the activation of GSR and TXNRD1 are involved in ROS elimination, if the LCS3-triggered lethality of VRK2-KO PC cells could be attributed to inhibiting the activity of both GSR and TXNRD1, then the level of ROS generated by LCS3 should have been much higher than those produced by the treatment of the TXNRD1 inhibitor auranofin

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