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Overactivation of PI3K/AKT induces premature activation of the dormant follicle pool and follicular burnout, culminating in fast depletion and eventual POF, whereas inhibition of PI3K/AKT may transpire in developing follicles, resulting in GC death and follicular atresia (Zhang et al

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This is akin to what happens in Graves disease, an autoimmune disorder where the bodys immune system mistakenly attacks the thyroid gland, leading to a cascade of health issues including rapid heartbeat, weight loss, and nervousness