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Cell lines stably expressing fluorescent-labeled AR, PR, GR and an internal control receptor(ICR)binding epitestosterone have been generated

Inhibition of NADPH oxidase-mediated superoxide radical formation in PMA-stimulated human neutrophils by 4-hydroxynonenal-binding to-SH and-NH2 groups

Moreover, ACTL6A overexpression relieved the inhibited GC cell proliferation caused by ACTL6A expression knockdown (Supplementary Fig 1c, d)