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Additionally, for non-Ca 2+ -permeable AMPA receptors, studies have shown that low-dose TNF- plays a neuroprotective role through tissue necrosis factor receptor-2 (TNFR-2), while high-dose TNF- increases the synaptic transport of calcium-permeable AMPARs through the TNFR-1 and enhances excitotoxicity in a concentration-dependent manner ( ) can exchange extracellular cystine with intracellular glutamic acid, thus providing the necessary raw materials for the synthesis of glutathione by glial cells

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(II) Despite the intracellular GSH being significantly consumed in the cases stated in this review, it is difficult to completely deplete GSH via these strategies